Diabetes & Obesity—Diabesity
A Genetic Curse or an Eating Hazard—How to Unmake the Mistake
by
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While in the fasting state, we do not need insulin to push the glucose into our brain cells. We do need some insulin to suppress lipolysis (to keep our fat stores from melting down). One of the main reasons overweight and obese individuals have a hard time losing weight is because every time they accumulate fat, the body will generate enough insulin to protect it and preserve it. This is the body defense for survival; it is a physiological response. As we accumulate more fat, more insulin is needed to protect against lipolysis, and the cycle goes on, and what starts as physiology goes on to pathology. The longer the duration of obesity and the degree of obesity, the more insulin is produced. This insulin, needed to protect the fat, is required constantly and not only briefly during and immediately after meals.
Simply said, instead of the ideal scenario, where the pancreatic beta cells are working three or four times every day to handle meals, with obesity they are working multiple shifts and overtime, day and night to suppress lipolysis, to protect fat stores in addition to handling meals. They are at risk for a burnout.
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About the Book
Diabesity is a growing nightmare. This book provides tips to balance the scale, and to help readers navigate their way out of this metabolic mess. It answers when, how, and what to eat. When, how, and for how long to exercise. It empowers readers to recognize the body’s worst metabolic enemy. That is visceral adiposity, or simply put fat deposition in the midsection.
For healthy non-diabetics, it is a call to action to avoid turning to a pre-diabetic, and to prevent those with pre-diabetes from progressing to diabetes.
For Diabetics, it presents the ambitious hope to reverse diabetes, or to slow its progression and prevent its devastating complications ranging from cardiovascular death to living blind, on dialysis or with an amputated limb.
It highlights in a storytelling mode the body intricate design to handle its metabolic demands. It then illustrates the linkage between the visceral fat deposition, and the body developing resistance to its own insulin. Insulin resistance is what leads eventually to the high blood pressure, the elevated blood glucose and abnormalities in the circulating lipids, namely cholesterol and similar particles, and even gout.
This is a tool in the hand of every one who wishes a healthier life, free from threats of Diabetes, obesity and their setbacks. It is the next right step after mere wishing.
About the Author
Dr. Helmy is an associate Professor of medicine at Indiana University. He is a triple board certified, internist, endocrinologist, and lipidologist, and an expert in his field. He is described by his colleagues and trainees as a consummate clinician and an exceptional educator of clinicians. He combines his clinical skills, and teacher talents, to present the latest scientific research. He is awarded multiple teaching awards, from Indiana University President award to the ACP laureate award, and many others.